📊 Classification
• Primary (SBP): haematogenous spread, no intra-abdominal source. Cirrhosis, nephrotic syndrome, post-necrotic ascites.
• Secondary: due to perforated viscus (appendicitis, diverticulitis, ulcer, trauma).
• Tertiary: persistent/recurrent after adequate therapy, often nosocomial pathogens.
• Peritoneal dialysis-related: from catheter contamination.
🦠 Microbiology — Primary (SBP)
• Monomicrobial (>90%)
• E. coli (40%), Klebsiella (7%), other GNB (20%)
• Streptococcus pneumoniae (15%), other Streptococcus (15%), Staph (3%)
• Gram-positive infections increasing
🦠 Microbiology — Secondary
• Depends on perforation level:
• Stomach: Lactobacillus, Candida
• Duodenum/proximal bowel: Bacteroides
• Colon: E. coli + Bacteroides fragilis (synergy)
📋 Diagnosis — SBP
• Ascitic fluid PMN >250 cells/mm³
• Culture positive
• pH <7.35, lactate >25 mg/dL
• Paracentesis: send for cell count, Gram stain, culture (inoculate blood culture bottles)
💊 Antibiotics — SBP
• 3rd gen cephalosporin (ceftriaxone/cefotaxime)
• Alternatives: ampicillin + aminoglycoside, piperacillin-tazobactam, meropenem
• Duration: 5 days (or longer based on response)
💊 Antibiotics — Secondary Peritonitis
• Aminoglycoside + clindamycin
• Or ceftriaxone + metronidazole
• Carbapenems or beta-lactam/beta-lactamase inhibitors for high-risk
• Surgical source control ESSENTIAL